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By Samuel K. Tan

A heritage of the Philippines, herein provided via Dr. Samuel okay. Tan … deals a conceptual framework of what he calls “the tale of guy within the Philippines” within the context of “the particular ecological system” and “distinctive historic experience” that experience formed his “particular personality and identity.” Dr. Tan offers during this narrow quantity an image of Philippine tradition which … “ought to be understood from the totality of the ethnolinguistic types which represent the material of Filipino society.” —Bernardita Reyes Churchill

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Maybe there were diets that reduced the amount of insulin released after a meal, which would mean that less fat was stored? I started looking and it wasn’t long before I stumbled onto GI diets. GI stands for glycaemic index and, simply put, it is a measure of the degree to which any given food produces a spike in blood sugar (and therefore insulin). It’s kind of like the different types of fuel you can feed a fire. If you put pine planks on the fire, they will burn very hot and very quickly but they will be used up in minutes.

Ghrelin is produced by the stomach lining in response to the presence of glucagon in the bloodstream, blocking the action of insulin (and leptin) in the hypothalamus. Circulating leptin levels stay reasonably constant most of the time, so our default appetite position is that we are full (or at least not hungry). Ghrelin counteracts the effect of leptin on the hypothalamus, effectively neutralising leptin for a while so we can feel hungry. The release of ghrelin is like throwing a ‘hungry’ switch in our brain.

Kojima and Kangawa found it in massive quantities in the stomach lining (in retrospect, not an entirely strange place to look for something that makes the tummy rumble at dinnertime). Ghrelin is produced by the stomach lining in response to the presence of glucagon in the bloodstream, blocking the action of insulin (and leptin) in the hypothalamus. Circulating leptin levels stay reasonably constant most of the time, so our default appetite position is that we are full (or at least not hungry). Ghrelin counteracts the effect of leptin on the hypothalamus, effectively neutralising leptin for a while so we can feel hungry.

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